Jan 20
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Rice:2008
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How does SLI develop later in childhood and into adulthood? Do those
with SLI eventually acquire the full grammar of the language, and if
so, is there a typical age at which the grammar is fully acquired?

My main takeaway from the Rice paper on SLI, is that children with SLI
exhibit largely the same language acquisition curve as unaffected
children, only on a delay (with some other confounding factors). My
question is whether or not being SLI affected as a child is predictive
of any disorders/deficits/issues in adulthood, or if it is merely a
delay of language acquisition.

The question of whether speech and language problems are related is a
little unclear to me. The author claims that they overlap, it turns
out that a child with speech problems probably has problems with
understanding and mastering the language?
   
The author also describes in detail the theory of asynchronous
language acquisition, which leads to a delay in speech. We discussed
that delays in speech and late language acquisition are typical
characteristic of bilingual children. Can they be attributed to this
delay theory?

I would be interested to know if there have been any breakthroughs or
further findings in the study of the genetic component of SLI since
the paper was published.

Why is nonword repetition an index of phonological short-term memory?

Initially, the FOXP2 gene was discussed greatly within the framework
of UG with researchers suggesting that this might be the "language
gene", the genetic manifestation of UG. Do genetics studies regarding
SLI belong also in the generative framework? The article mentions two
chromosomes (19q, 19q). Are those linked explicitly to language
abilities or do they have ties to more general cognitive functions?

What advantages do affected children have over unaffected children in
terms of language acquisition?

How would deviant language acquisition look like?

When speaking of SLI, based off the authors' description as well as
the experimental settings it seems they isolate it to spoken
impairment- so production of speech. Do they also consider impairment
in comprehension + actually processing of linguistic data? I would
guess those things are quite hard to isolate from speech production.